[et_pb_section bb_built=”1″][et_pb_row][et_pb_column type=”4_4″][et_pb_text _builder_version=”3.13.1″]

To know in good faith, about the science of cancer tumors and what establishes their development, a group of scientists from CIBERONC, in Spain, have discovered that patients suffering from cancer of the colon phospholipase D2 protein, have irregularly high levels, which is produced by the tumor cells and changes to non-tumor cells causing it, which leads to tumor growth.

When performing an analysis of healthy and damaged cells around the environment, many indicators contribute to the spread of cancer. However, this could be the way to establish the causes of the disease.

According to the scientists of the CIBERONC, the cancer cells are in continuous communication with the microenvironment and can affect the evolution of the tumor. “We have observed that the expression of protein phospholipase D2 increases in colorectal cancer, which induces senescence in the fibroblasts of adjacent cells and leads to a greater proliferative capacity than normal tumor cells, increasing resistance to antitumor treatments.

The fibroblasts are responsible for producing the extracellular matrix and collagen necessary for animal tissues. The main function of defibroblasts is the maintenance of structural integrity within the connective tissue.

Senescence is the process initiated in response to stress and damage occurring in a cell, and establishes an alternative response to programmed cell death and is of vital importance to suppress the formation of cancerous tumor cells.

These and other innovations are now possible in Pharmamedic.

[/et_pb_text][/et_pb_column][/et_pb_row][/et_pb_section]